Shaw, SG

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Journal Article

Simic, B; Hermann, M; Shaw, SG; Bigler, L; Stalder, U; Dorries, C; Besler, C; Luscher, TF; Ruschitzka, F (2012). Torcetrapib impairs endothelial function in hypertension. European Heart Journal, 33(13), pp. 1615-1624. Oxford: Oxford University Press 10.1093/eurheartj/ehr348

Rose, AK; Shaw, SG; Prendergast, MA; Little, HJ (2010). The Importance of Glucocorticoids in AlcoholDependence and Neurotoxicity. Alcoholism: clinical and experimental research, 34(12), pp. 2011-2018. Oxford: Wiley-Blackwell 10.1111/j.1530-0277.2010.01298.x

Shaw, SG; Boden, PJ (2005). Insulin resistance, obesity and the metabolic syndrome. Is there a therapeutic role for endothelin-1 antagonists? Current vascular pharmacology, 3(4), 359-63.. San Francisco, Calif.: Bentham Science Publishers

Koshy, A; De Gottardi, A; Ledermann, M; Saegesser, H; Shaw, SG; Zimmermann, A; Reichen, J (2005). Endothelial nitric oxide synthase is not essential for the development of fibrosis and portal hypertension in bile duct ligated mice. Liver international, 25(5), 1044-52.. Oxford: Blackwell Munksgaard 10.1111/j.1478-3231.2005.01146.x

Emre, M; Orgul, S; Haufschild, T; Shaw, SG; Flammer, J (2005). Increased plasma endothelin-1 levels in patients with progressive open angle glaucoma. British journal of ophthalmology, 89(1), 60-3.. London: BMJ Publishing Group 10.1136/bjo.2004.046755

Ferrari, P; Shaw, SG; Nicod, J; Saner, E; Nussberger, J (2004). Active renin versus plasma renin activity to define aldosterone-to-renin ratio for primary aldosteronism. Journal of hypertension, 22(2), 377-81.. Hagerstown, Md.: Lippincott Williams & Wilkins 10.1097/01.hjh.0000098195.36890.47

Haufschild, T; Shaw, SG; Kaiser, HJ; Flammer, J (2003). Transient raise of endothelin-1 plasma level and reduction of ocular blood flow in a patient with optic neuritis. Ophthalmologica, 217(6), 451-3.. Basel: Karger 10.1159/000073079

Balsiger, B; Rickenbacher, A; Boden, PJ; Biecker, E; Tsui, J; Dashwood, M; Reichen, J; Shaw, SG (2002). Endothelin A-receptor blockade in experimental diabetes improves glucose balance and gastrointestinal function. Clinical science, 48(103), 430S-433S.. London: Portland

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